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- A left anterior oblique view of the right coronary artery after percutaneous intervention.
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- Ajayi AAL, Mathur R, Halushka PV. Testosterone enhance human platelet thromboxane A2 receptor density and aggregation responses.
- The left anterior artery demonstrates a 95% stenosis after the first septal branch.
We report a case of an acute ST-segment elevation myocardial infarction in a affected person with conventional cardiac danger components utilizing supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person also had polycythemia, likely secondary to high-dose testosterone. The patient underwent successful percutaneous intervention of the proper coronary artery. Physicians and patients should be aware of the medical penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement therapy and recommendations for monitoring. Testosterone replacement in hypogonadal males with angina improves ischaemic threshold and high quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone improve human platelet thromboxane A2 receptor density and aggregation responses.
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Moreover, a part of endothelial dysfunction has been proposed, which can contribute to irregular vessel reactivity. However, testosterone in physiologic doses might even be useful in sufferers with angina , have larger ischemic thresholds and improved quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and will increase coronary blood move in men with established coronary artery disease . Therefore, an interplay of endogenous components of the patient’s risk profile in addition to the testosterone dosing may be issues of whether adverse occasions happen.
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